Anatomy of the cns

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  • The brain and spinal cord compose the Central Nervous System (CNS), which is the control center of the body. Inputs from muscles, involuntary organs, and senses travel through the nerves of the Peripheral Nervous System (PNS) into the CNS where they are interpreted. Signals may travel within the brain to separate functional areas. Instructions are then sent outward again for voluntary movement and involuntary regulation to complete the endless loop of the nervous system circuitry.

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  • Hop up, Dick, love! See how glorious the sun is on the new snow. Now isn't that more beautiful than your dreams? And see the birdies! They can't find any breakfast. Let's hurry and have our morning wrestle and dress and give them some breakie before Anne calls." The mother is Ethel Baxter Lord. She is thirty-eight, and Dick-boy is just five. The mother's face is striking, striking as an example of fine chiseling of features, each line standing for sensitiveness, and each change revealing refinement of thought. The eyes and hair are richly brown.

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  • Chapter 12 - The central nervous system (part a), the topics discussed in this chapter are: Central Nervous System (CNS), embryonic development, effect of space restriction on brain development, regions and organization of the CNS, ventricles of the brain,...and other contents.

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  • Herpes simplex viruses (HSV-1, HSV-2; Herpesvirus hominis) produce a variety of infections involving mucocutaneous surfaces, the central nervous system (CNS), and—on occasion—visceral organs. Prompt recognition and treatment reduce the morbidity and mortality associated with HSV infections. Etiologic Agent The genome of HSV is a linear, double-strand DNA molecule (molecular weight, ~100 x 106 units) that encodes 90 transcription units with 84 identified proteins. The genomic structures of the two HSV subtypes are similar.

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  • Syphilis, a chronic systemic infection caused by Treponema pallidum subspecies pallidum, is usually sexually transmitted and is characterized by episodes of active disease interrupted by periods of latency. After an incubation period averaging 2–6 weeks, a primary lesion appears, often associated with regional lymphadenopathy. The secondary stage, associated with generalized mucocutaneous lesions and generalized lymphadenopathy, is followed by a latent period of subclinical infection lasting years or decades.

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