Xem 1-19 trên 19 kết quả Blisters
  • Tuyển tập các báo cáo nghiên cứu về y học được đăng trên tạp chí y học quốc tế cung cấp cho các bạn kiến thức về ngành y đề tài: Matrix-metalloproteinase-2, -8 and -9 in serum and skin blister fluid in patients with severe sepsis...

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  • Table 54-12 Causes of Vesicles/Bullae I. Primary cutaneous diseases A. Primary blistering diseases (autoimmune) 1. Pemphigusa 2. Bullous pemphigoidb 3. Gestational pemphigoidb 4. Cicatricial pemphigoidb 5. Dermatitis herpetiformisb,c 6. Linear IgA bullous dermatosisb 7. Epidermolysis bullosa acquisitab,d B. Secondary blistering diseases 1. Contact dermatitisa 2. Erythema multiformea,b 3. Stevens-Johnson syndrome 4. Toxic epidermal necrolysisb C. Infections 1. Varicella/zoster virusa,e 2. Herpes simplex virusa,e 3. Enteroviruses, e.g., hand-foot-and-mouth disease 4.

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  • Móng tay giòn mỹ phẩm điều trị bệnh teo móng tay paronychia cấp Blistering viêm ngón ngoại biên mãn tính paronychia Onycholysis Bệnh vẩy nến liken phẳng Hai mươi loạn dưỡng móng tay vàng Onychogryphosis hội chứng móng tay móng cắn và mụn cóc onychotillomania Periungual

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  • Several metabolic disorders are associated with blister formation, including diabetes mellitus, renal failure, and porphyria. Local hypoxia secondary to decreased cutaneous blood flow can also produce blisters, which explains the presence of bullae over pressure points in comatose patients (coma bullae). In diabetes mellitus, tense bullae with clear viscous fluid arise on normal skin. The lesions can be as large as 6 cm in diameter and are located on the distal extremities. There are several types of porphyria, but the most common form with cutaneous findings is PCT.

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  • HFMD is a common viral illness among infants and young children. This virus can cause fever and sores in the mouth, and blisters on the hands and feet. The disease is usually mild but it can also cause severe condition, complications and sometimes results in death. The virus causing HFMD is spread from person to person through direct contact through nose and throat secretions, saliva, blister fluids, stools of infected persons, or through contact with contaminated surfaces.

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  • More serious rusting can lead to obvious holes in the metalwork, or as often happens in double skinned areas such as rear wheelarches or door bottoms, rust or blisters which appear because the panel has rusted quietly away from the inside. By the time the first signs show, the metal in that area is already beyond help. The only long term repair will involve a panel\section replacement, or if the area is very small and the rest of the panel is absolutely sound, body solder can be used effectively. For a purely cosmetic temporary repair, the affected area can...

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  • Pemphigus Vulgaris Pemphigus refers to a group of autoantibody-mediated intraepidermal blistering diseases characterized by loss of cohesion between epidermal cells (a process termed acantholysis). Manual pressure to the skin of these patients may elicit the separation of the epidermis (Nikolsky's sign). This finding, while characteristic of pemphigus, is not specific to this group of disorders and is also seen in toxic epidermal necrolysis, Stevens-Johnson syndrome, and a few other skin diseases.

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  • Pemphigus Foliaceus Pemphigus foliaceus (PF) is distinguished from PV by several features. In PF, acantholytic blisters are located high within the epidermis, usually just beneath the stratum corneum. Hence PF is a more superficial blistering disease than PV. The distribution of lesions in the two disorders is much the same, except that in PF mucous membranes are almost always spared. Patients with PF rarely demonstrate intact blisters but rather exhibit shallow erosions associated with erythema, scale, and crust formation.

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  • These reactions are characterized by one or more sharply demarcated, erythematous lesions, sometimes leading to a blister. Hyperpigmentation results after resolution of the acute inflammation. With rechallenge, the lesion recurs in the same (i.e., fixed) location. Lesions often involve the lips, hands, legs, face, genitalia, and oral mucosa and cause a burning sensation. Most patients have multiple lesions. Fixed drug eruptions have been associated with phenolphthalein, sulfonamides, cyclines, dipyrone, NSAIDs, and barbiturates.

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  • Skin and Soft Tissue Infections S. aureus causes a variety of cutaneous infections. Common predisposing factors include skin disease, skin damage (e.g., insect bites, minor trauma), injections (e.g., in diabetes, injection drug use), and poor personal hygiene. These infections are characterized by the formation of pus-containing blisters, which often begin in hair follicles and spread to adjoining tissues. Folliculitis is a superficial infection that involves the hair follicle, with a central area of purulence (pus) surrounded by induration and erythema.

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  • The remit for the first edition of the ABC of Dermatology in 1987 was that it should concentrate on common conditions and give down to earth advice. The ABC format proved well suited for this and there has been a steady demand for the book since then. In this edition the same approach is maintained while taking into account advances in diagnosis and treatment. Research in genetics and immunology is providing ever-increasing insights into the mechanisms that underlie clinical changes, and has led to more accurate diagnosis and more rational treatment.

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  • The Contagious Distemper, which has become the Reproach of our Faculty here for above a Month past, is more violent than that at Marseilles; it breaks out in Carbuncles, Buboes, livid Blisters, and purple Spots; the first Symptoms are grievous Pains in the Head, Consternations, wild Looks, a trembling Voice, a cadaverous Face, a Coldness in all the extreme Parts, a low unequal Pulse, great Pains in the Stomach, Reachings to Vomit, and these are follow'd by Sleepiness, Deliriums, Convulsions, or Fluxes of Blood, the Forerunners of sudden Death.

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  • The ChillTip handpiece must be in firm contact with the skin. A single pulse should be placed at test sites within or near the treatment area. If epidermal damage is present (blistering, abla- tion, graying or whitening of the epidermis, or a positive Nikolski sign) the fluence should be lowered by 5 to 10 J/cm2 . Several pulses should then be placed next to one another while looking for the epidermal response. An effective fluence is one where the hair carbonizes, followed by very selective follicular swelling and redness. ...

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  • ON DECEMBER 7, 1940, DURING THE SEASON FINALE AT SOUTHERN California, Notre Dame coach Elmer Layden charged onto the field to protest what he felt was a rotten call. But Layden didn’t stop there. After blistering the refs, he screamed at USC coach Howard Jones. A normally genial man, Layden had finally submitted to the abnormal pressure of coaching Notre Dame football. This pressure had increased for nine straight years—ever since March 31, 1931, the stunning day Knute Rockne died in an airplane crash....

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  • Bullous Pemphigoid Bullous pemphigoid (BP) is a polymorphic autoimmune subepidermal blistering disease usually seen in the elderly. Initial lesions may consist of urticarial plaques; most patients eventually display tense blisters on either normalappearing or erythematous skin (Fig. 55-2). The lesions are usually distributed over the lower abdomen, groin, and flexor surface of the extremities; oral mucosal lesions are found in some patients. Pruritus may be nonexistent or severe.

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  • Pemphigoid Gestationis Pemphigoid gestationis (PG), also known as herpes gestationis, is a rare, nonviral, subepidermal blistering disease of pregnancy and the puerperium. PG may begin during any trimester of pregnancy or present shortly after delivery. Lesions are usually distributed over the abdomen, trunk, and extremities; mucous membrane lesions are rare. Skin lesions in these patients may be quite polymorphic and consist of erythematous urticarial papules and plaques, vesiculopapules, and/or frank bullae. Lesions are almost always very pruritic.

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  • Linear IgA Disease Linear IgA disease, once considered a variant form of dermatitis herpetiformis, is actually a separate and distinct entity. Clinically, these patients may resemble individuals with DH, BP, or other subepidermal blistering diseases. Lesions typically consist of papulovesicles, bullae, and/or urticarial plaques predominantly on central or flexural sites. Oral mucosal involvement occurs in some patients. Severe pruritus resembles that seen in patients with DH.

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  • There are two situations in which the TST is not required. Applicants providing written documentation (with a health-care provider’s signature) of a TST reaction of 5 mm or greater of induration or applicants with a history of a severe reaction with blistering to a prior TST may be excluded from this requirement. Applicants in these two groups must undergo a chest radiograph. A verbal history of a positive TST reaction from the applicant is not acceptable.

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  • Bài giảng Phân loại mô học các bệnh bóng nước của BS. Hà Văn Phước trình bày về đặc điểm, phân loại bệnh bóng nước; bóng nước dưới lớp sừng (subcorneal blister); bóng nước do thoái hóa nội bào (intracellular degeneration); bóng nước xốp bào (spongiotic blister); bóng nước có tiêu gai (acantholytic blister) cùng một số bệnh bóng nước khác, mời các bạn tham khảo.

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