Endocrinology

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  • During the last two decades, evidence of increasing trends of several endocrine-related disorders has been strengthened. These disorders often come with lack of uniform diagnosis and/ or even unclear endocrine disruption. The later is mainly due to abnormal classical changes in the blood- released hormone to its targeted organ, abnormal communication between cells within a tissue or organ (paracrine), within the same cell (intracrine) or signals which act on the same cell (autocrine).

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  • Harrison's Internal Medicine Chapter 96. Paraneoplastic Syndromes: Endocrinologic/Hematologic Paraneoplastic Syndromes: Endocrinologic/Hematologic: Introduction In addition to local tissue invasion and metastasis, neoplastic cells can produce a variety of peptides that that can stimulate hormonal, hematologic, dermatologic, or neurologic responses. Paraneoplastic syndromes refer to the disorders that accompany benign or malignant tumors but are not directly related to mass effects or invasion.

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  • Endocrinology: Basic and Clinical Principles, Second Edition aims to provide a comprehensive knowledge base for the applied and clinical science of endocrinology. The challenge in its presentation was to produce a volume that was timely, provided integration of basic science with physiologic and clinical principles, and yet was limited to 500 pages. This length makes the volume suitable as a text; and the timeliness we have striven for allows the book to serve as an offthe- shelf reference.

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  • Nearly a decade elapsed between publication of the second and third editions of Basic Medical Endocrinology due in large part to the turmoil in the publishing industry brought on by massive consolidation.Although this edition is new and the publisher is new, the aims of earlier editions of this work are unchanged. Its focus remains human endocrinology with an emphasis on cellular and molecular mechanisms presented in the context of integration of body functions.

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  • e body of knowledge in endocrinology has expanded enormously since the fi rst edition of this text appeared two decades ago, and the pace of discovery has been no less robust since the appearance of the third edition. Research in endocrinology continues to produce new revelations and insights, sometimes deepening our level of understanding of well-established phenomena, and sometimes leading us to reevaluate and reinterpret long-held doctrines.

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  • The aim of the Handbook of Diagnostic Endocrinology is to provide a comprehensive overview of current approaches to the diagnosis of endocrine disorders. Our ability to diagnose patients with diseases of the endocrine systems is expanding exponentially with the development of new and more reliable assay methods and the incorporation of both molecular and genetic approaches into our understanding of the pathophysiology of these diseases.

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  • (BQ) Nối tiếp nội dung của phần 1 cuốn sách, phần 2 trình bày các nội dung: Disorders of the Kidney and Urinary Tract, Disorders of the Gastrointestinal System, Rheumatology and Immunology, Endocrinology and Matabolism, Neurologic Disorders, dermatology. Mời các bạn cùng tham khảo nội dung chi tiết.

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  • Tham khảo sách 'basic medical endocrinology - third edition', khoa học tự nhiên, công nghệ sinh học phục vụ nhu cầu học tập, nghiên cứu và làm việc hiệu quả

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  • This textbook is primarily intended to provide undergraduate students of pharmacy with a clear and concise account of basic endocrine function and dysfunction, at a level sufficient to meet the requirements of first- or second year qualifying examinations. It is not intended to replace standard texts, but merely to serve as an accompaniment and convenient revision guide.

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  • Ectopic hormone production would only be an epiphenomenon associated with cancer if it did not result in clinical manifestations. Excessive and unregulated production of hormones such as ACTH, PTHrP, or vasopressin can lead to substantial morbidity and can complicate the cancer treatment plan. Moreover, the paraneoplastic endocrinopathies are sometimes the presenting feature of underlying malignancy and may prompt the search for an unrecognized tumor.

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  • Tumor-Induced Hypoglycemia Caused by Excess Production of IGF-II (See also Chap. 339) Mesenchymal tumors, hemangiopericytomas, hepatocellular tumors, adrenal carcinomas, and a variety of other large tumors have been reported to produce excessive amounts of insulin-like growth factor type II (IGF-II) precursor, which binds weakly to insulin receptors and strongly to IGF-I receptors, leading to insulin-like actions. The gene encoding IGF-II resides on a chromosome 11p15 locus that is normally imprinted (that is, expression is exclusively from a single parental allele).

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  • Hematologic Syndromes: Introduction The elevation of granulocyte, platelet, and eosinophil counts in most patients with myeloproliferative disorders is caused by the proliferation of the myeloid elements due to the underlying disease rather than a paraneoplastic syndrome. The paraneoplastic hematologic syndromes in patients with solid tumors are less well characterized than the endocrine syndromes because the ectopic hormone(s) or cytokines responsible have not been identified in most of these tumors (Table 96-2).

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  • Granulocytosis Approximately 30% of patients with solid tumors have granulocytosis (granulocyte count 8000/µL). In about half of patients with granulocytosis and cancer, the granulocytosis has an identifiable nonparaneoplastic etiology (infection, tumor necrosis, glucocorticoid administration, etc.). The other patients have proteins in urine and serum that stimulate the growth of bone marrow cells.

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  • Clinical Manifestations Patients with cancer who develop deep venous thrombosis usually develop swelling or pain in the leg, and physical examination reveals tenderness, warmth, and redness. Patients who present with pulmonary embolism develop dyspnea, chest pain, and syncope, and physical examination shows tachycardia, cyanosis, and hypotension. Some 5% of patients with no history of cancer who have a diagnosis of deep venous thrombosis or pulmonary embolism will have a diagnosis of cancer within 1 year.

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  • Recent expansion of biomedical knowledge on the interactions between the fetus, placenta, and the mother have transformed our view of pregnancy in general. Recent basic and clinical investigations have improved significantly our understanding on how hormones affect the pregnancy, and on how pregnancy affects the fetal and maternal hormones. Because pregnancy may be seen as the ultimate hormonally mediated event, the topic of endocrinology of pregnancy is particularly relevant.

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  • Etiology Humoral hypercalcemia of malignancy (HHM) occurs in up to 20% of patients with cancer. HHM is most common in cancers of the lung, head and neck, skin, esophagus, breast, genitourinary tract, and in multiple myeloma and lymphomas. Several distinct humoral causes of HHM occur, most commonly overproduction of PTHrP. In addition to acting as a circulating humoral factor, bone metastases (e.g., breast, multiple myeloma) may produce PTHrP, leading to local osteolysis and hypercalcemia.

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  • Etiology Vasopressin is an antidiuretic hormone normally produced by the posterior pituitary gland. Ectopic vasopressin production by tumors is a common cause of the syndrome of inappropriate antidiuretic hormone (SIADH), occurring in at least half of patients with SCLC. Compensatory mechanisms, such as decreased thirst, suppression of aldosterone, and production of atrial natriuretic peptide (ANP), may mitigate the development of hyponatremia in patients who produce excessive vasopressin.

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  • Rarely, corticotropin-releasing hormone (CRH) is produced by pancreatic islet tumors, SCLC, medullary thyroid cancer, carcinoids, or prostate cancer. When levels are high enough, CRH can cause pituitary corticotrope hyperplasia and Cushing's syndrome. Tumors that produce CRH sometimes also produce ACTH, raising the possibility of a paracrine mechanism for ACTH production. A distinct mechanism for ACTH-independent Cushing's syndrome involves ectopic expression of various G protein–coupled receptors in the adrenal nodules.

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  • Tuyển tập các báo cáo nghiên cứu về y học được đăng trên tạp chí y học quốc tế cung cấp cho các bạn kiến thức về ngành y đề tài: Identification and isolation of embryonic stem cells in reproductive endocrinology: theoretical protocols for conservation of human embryos derived from in vitro fertilization

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  • Chapter 26 - Endocrinology, this chapter describe how hormones secreted from the endocrine glands help body maintain homeostasis; describe anatomy and physiology of pancreas and how its hormones maintain normal glucose metabolism; discuss pathophysiology as a basis for key signs and symptoms, patient assessment, and patient management for diabetes and diabetic emergencies of hypoglycemia, diabetic ketoacidosis, and hyperosmolar hyperglycemic nonketotic syndrome;...

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