Endocrinology

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  • During the last two decades, evidence of increasing trends of several endocrine-related disorders has been strengthened. These disorders often come with lack of uniform diagnosis and/ or even unclear endocrine disruption. The later is mainly due to abnormal classical changes in the blood- released hormone to its targeted organ, abnormal communication between cells within a tissue or organ (paracrine), within the same cell (intracrine) or signals which act on the same cell (autocrine).

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  • Harrison's Internal Medicine Chapter 96. Paraneoplastic Syndromes: Endocrinologic/Hematologic Paraneoplastic Syndromes: Endocrinologic/Hematologic: Introduction In addition to local tissue invasion and metastasis, neoplastic cells can produce a variety of peptides that that can stimulate hormonal, hematologic, dermatologic, or neurologic responses. Paraneoplastic syndromes refer to the disorders that accompany benign or malignant tumors but are not directly related to mass effects or invasion.

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  • (BQ) Part 2 book "Harrison's endocrinology" presents the following contents: Diabetes mellitus, obesity, lipoprotein metabolism; disorders affecting multiple endocrine systems; disorders of bone and calcium metabolism; laboratory values of clinical importance.

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  • (BQ) Part 1 book "Harrison's endocrinology" presents the following contents: Principles of endocrinology; pituitary, thyroid, and adrenal disorders; reproductive endocrinology. Invite you to consult.

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  • (BQ) Part 1 book "Oxford handbook of endocrinology and diabetes" presents the following contents: Thyroid, pituitary, aituitaryl, reproductive endocrinology. Invite you to consult.

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  • (BQ) Part 2 book "Oxford handbook of endocrinology and diabetes" presents the following contents: Endocrinology in pregnancy, calcium and bone metabolism, paediatric endocrinology, neuroendocrine disorders, inherited endocrine syndromes and MEN, endocrine surgery,... and other contents.

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  • Endocrinology: Basic and Clinical Principles, Second Edition aims to provide a comprehensive knowledge base for the applied and clinical science of endocrinology. The challenge in its presentation was to produce a volume that was timely, provided integration of basic science with physiologic and clinical principles, and yet was limited to 500 pages. This length makes the volume suitable as a text; and the timeliness we have striven for allows the book to serve as an offthe- shelf reference.

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  • Nearly a decade elapsed between publication of the second and third editions of Basic Medical Endocrinology due in large part to the turmoil in the publishing industry brought on by massive consolidation.Although this edition is new and the publisher is new, the aims of earlier editions of this work are unchanged. Its focus remains human endocrinology with an emphasis on cellular and molecular mechanisms presented in the context of integration of body functions.

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  • e body of knowledge in endocrinology has expanded enormously since the fi rst edition of this text appeared two decades ago, and the pace of discovery has been no less robust since the appearance of the third edition. Research in endocrinology continues to produce new revelations and insights, sometimes deepening our level of understanding of well-established phenomena, and sometimes leading us to reevaluate and reinterpret long-held doctrines.

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  • The aim of the Handbook of Diagnostic Endocrinology is to provide a comprehensive overview of current approaches to the diagnosis of endocrine disorders. Our ability to diagnose patients with diseases of the endocrine systems is expanding exponentially with the development of new and more reliable assay methods and the incorporation of both molecular and genetic approaches into our understanding of the pathophysiology of these diseases.

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  • (BQ) Nối tiếp nội dung của phần 1 cuốn sách, phần 2 trình bày các nội dung: Disorders of the Kidney and Urinary Tract, Disorders of the Gastrointestinal System, Rheumatology and Immunology, Endocrinology and Matabolism, Neurologic Disorders, dermatology. Mời các bạn cùng tham khảo nội dung chi tiết.

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  • Ectopic hormone production would only be an epiphenomenon associated with cancer if it did not result in clinical manifestations. Excessive and unregulated production of hormones such as ACTH, PTHrP, or vasopressin can lead to substantial morbidity and can complicate the cancer treatment plan. Moreover, the paraneoplastic endocrinopathies are sometimes the presenting feature of underlying malignancy and may prompt the search for an unrecognized tumor.

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  • Tumor-Induced Hypoglycemia Caused by Excess Production of IGF-II (See also Chap. 339) Mesenchymal tumors, hemangiopericytomas, hepatocellular tumors, adrenal carcinomas, and a variety of other large tumors have been reported to produce excessive amounts of insulin-like growth factor type II (IGF-II) precursor, which binds weakly to insulin receptors and strongly to IGF-I receptors, leading to insulin-like actions. The gene encoding IGF-II resides on a chromosome 11p15 locus that is normally imprinted (that is, expression is exclusively from a single parental allele).

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  • Hematologic Syndromes: Introduction The elevation of granulocyte, platelet, and eosinophil counts in most patients with myeloproliferative disorders is caused by the proliferation of the myeloid elements due to the underlying disease rather than a paraneoplastic syndrome. The paraneoplastic hematologic syndromes in patients with solid tumors are less well characterized than the endocrine syndromes because the ectopic hormone(s) or cytokines responsible have not been identified in most of these tumors (Table 96-2).

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  • Granulocytosis Approximately 30% of patients with solid tumors have granulocytosis (granulocyte count 8000/µL). In about half of patients with granulocytosis and cancer, the granulocytosis has an identifiable nonparaneoplastic etiology (infection, tumor necrosis, glucocorticoid administration, etc.). The other patients have proteins in urine and serum that stimulate the growth of bone marrow cells.

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  • Clinical Manifestations Patients with cancer who develop deep venous thrombosis usually develop swelling or pain in the leg, and physical examination reveals tenderness, warmth, and redness. Patients who present with pulmonary embolism develop dyspnea, chest pain, and syncope, and physical examination shows tachycardia, cyanosis, and hypotension. Some 5% of patients with no history of cancer who have a diagnosis of deep venous thrombosis or pulmonary embolism will have a diagnosis of cancer within 1 year.

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  • Tham khảo sách 'basic medical endocrinology - third edition', khoa học tự nhiên, công nghệ sinh học phục vụ nhu cầu học tập, nghiên cứu và làm việc hiệu quả

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  • This textbook is primarily intended to provide undergraduate students of pharmacy with a clear and concise account of basic endocrine function and dysfunction, at a level sufficient to meet the requirements of first- or second year qualifying examinations. It is not intended to replace standard texts, but merely to serve as an accompaniment and convenient revision guide.

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  • (BQ) Part 2 book "BRS biochemistry molecular biology and genetics" presents the following contents: Lipid and ethanol metabolism; nitrogen metabolism–amino acids, purines, pyrimidines and products derived from amino acids; molecular endocrinology and an overview of tissue metabolism, human genetics—an introduction.

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  • Etiology Humoral hypercalcemia of malignancy (HHM) occurs in up to 20% of patients with cancer. HHM is most common in cancers of the lung, head and neck, skin, esophagus, breast, genitourinary tract, and in multiple myeloma and lymphomas. Several distinct humoral causes of HHM occur, most commonly overproduction of PTHrP. In addition to acting as a circulating humoral factor, bone metastases (e.g., breast, multiple myeloma) may produce PTHrP, leading to local osteolysis and hypercalcemia.

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