Xem 1-8 trên 8 kết quả Estrogen therapy
  • Calcified plaque in the coronary arteries is a marker for atheromatous-plaque burden and is predictive of future risk of cardiovascular events. We examined the relationship between estrogen therapy and coronary-artery calcium in the context of a randomized clinical trial. METHODS In our ancillary substudy of the Women’s Health Initiative trial of conjugated equine estrogens (0.625 mg per day) as compared with placebo in women who had undergone hysterectomy, we performed computed tomography of the heart in 1064 women aged 50 to 59 years at randomization.

    pdf13p muakhuya 07-07-2012 28 1   Download

  • THỬ NGHIỆM HOẠT TÍNH ESTROGEN CỦA MẦM HẠT ĐẬU NÀNH TÓM TẮT Mục tiêu: Hạt Đậu nành (Glycine max) là nguồn nguyên liệu chứa nhiều genistein, đây là một loại isoflavonoid có hoạt tính estrogen, được dùng như một liệu pháp hormon thay thế HRT (hormone replacement therapy). Những thử nghiệm dược lý đã chứng minh các flavonoid trong mầm hạt Đậu nành có thể hỗ trợ cho các trường hợp thiểu năng estradiol trên chuột thí nghiệm.

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  • The hippocampus and cerebral cortex are areas associated with seizure initiation and propagation. It is therefore discussed how pathological disturbances in the local estrogen production after menopause may contribute to an increase in seizures in some women (Veliskova, 2007). Estrone is the primary estrogen after menopause, and its main source is subcutaneous fat. This might be of importance for overweight women with epilepsy. Little is known on the influence of estrone on epilepsy.

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  • Estrogen therapy: Using estrogen to treat symptoms of change of life is known as estrogen therapy or menopausal hormone therapy. Estrogen treatment can reduce hot flashes, improve vaginal dryness, and help prevent the weakening of the bones (osteoporosis) that can happen with menopause. But the use of estrogen by itself increases a woman's risk of getting endometrial cancer in women who still have a uterus. Estrogen therapy also increases a woman's chance of developing serious blood clots and heart disease.

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  • Endocrine Therapy Normal breast tissue is estrogen-dependent. Both primary and metastatic breast cancer may retain this phenotype. The best means of ascertaining whether a breast cancer is hormone-dependent is through analysis of estrogen and progesterone receptor levels on the tumor. Tumors that are positive for the estrogen receptor and negative for the progesterone receptor have a response rate of ~30%. Tumors that have both receptors have a response rate approaching 70%. If neither receptor is present, the objective response rates are ...

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  • Chemoprevention of Breast Cancer Hormonal manipulation is being tested in the primary prevention of breast cancer. Tamoxifen is an antiestrogen with partial estrogen agonistic activity in some tissues, such as endometrium and bone. One of its actions is to upregulate transforming growth factor β, which decreases breast cell proliferation. In randomized placebo-controlled trials to assess tamoxifen as adjuvant therapy for breast cancer, tamoxifen reduced the number of new breast cancers in the opposite breast by more than a third.

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  • • Menopausal hormone therapy: Some studies have suggested that women who take estrogen by itself (estrogen without progesterone) for 10 or more years may have an increased risk of ovarian cancer. Scientists have also studied whether taking certain fertility drugs, using talcum powder, or being obese are risk factors. It is not clear whether these are risk factors, but if they are, they are not strong risk factors. Having a risk factor does not mean that a woman will get ovarian cancer. Most women who have risk factors do not get ovarian cancer.

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  • Do the cancer cells have hormone receptors? Hormone receptors are like ears on and in breast cells that listen to signals from hormones. These hormone signals tell breast cells that have the receptors to grow. A cancer is called eR-positive if it has receptors for the hormone estrogen. It’s called PR-positive if it has receptors for the hormone progesterone. Breast cells that do not have receptors are negative for these hormones. Breast cancers that are ER-positive, PR-positive, or both tend to respond to hormonal therapy.

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