Neuromuscular physiology

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  • (BQ) Part 1 book "Medical physiology principles for clinical medicine" presents the following contents: Cellular physiology, neuromuscular physiology, blood and immunology, cardiovascular physiology.

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  • T his book seeks to provide a comprehensive orientation to the fundamental concepts and clinical applications of Muscle Energy Technique (MET) as they have evolved over more than forty years. Fred L. Mitchell, Sr., DO, FAAO, the original developer of Muscle Energy Technique, never wrote a book, or even a paper, about MET specifically. Mitchell, Sr. did, however, write two highly significant papers! (1948, 1958) about manipulative treatment of mechanical dysfunctions of the pelvic joints. He gave credit to T.J. Ruddy, D.O. (1874-1964) and Carl Kettler, D.O.

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  • Acetylcholine is a widespread chemotransmitter in the body, mediating a broad range of physiological effects.There are two distinct classes of receptor for acetylcholine defined on the basis of their preferential activation by the alkaloids, nicotine (from tobacco) and muscarine (from a fungus, Amanita muscaria). Cholinergic drugs (acetylcholine agonists) mimic acetylcholine at all sites although the balance of nicotinic and muscarinic effects is variable.

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  • Harrison's Hypocalcemia Internal Medicine Chapter 47. Hypercalcemia and HYPERCALCEMIA AND HYPOCALCEMIA: INTRODUCTION The calcium ion plays a critical role in normal cellular function and signaling, regulating diverse physiologic processes such as neuromuscular signaling, cardiac contractility, hormone secretion, and blood coagulation. Thus, extracellular calcium concentrations are maintained within an exquisitely narrow range through a series of feedback mechanisms that involve parathyroid hormone (PTH) and the active vitamin D metabolite 1,25-dihydroxyvitmin D [1,25(OH)2D].

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  • The calcium ion plays a critical role in normal cellular function and signaling, regulating diverse physiologic processes such as neuromuscular signaling, cardiac contractility, hormone secretion, and blood coagulation. Thus, extracellular calcium concentrations are maintained within an exquisitely narrow range through a series of feedback mechanisms that involve parathyroid hormone (PTH) and the active vitamin D metabolite 1,25-dihydroxyvitmin D [1,25(OH)2D]. These feedback mechanisms are orchestrated by integrating signals between the parathyroid glands, kidney, intestine, and bone (Fig.

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