CO2 injection has recently been shown to provide significant enhanced oil
recovery from naturally fractured reservoirs1. Li et al. (2000) performed CO2
injection at 1750 psig on artificially-fractured cores after water flooding in a
dead-oil system. Gravity drainage was suggested to be the dominant recovery
mechanism in these tests, with significant tertiary oil recovery after water
flooding. The authors observed that the recovery of initial oil at the start of the
CO2 injection declined as the rock permeability decreased and the initial water
A clear understanding of rock stress and its effect on permeability is important in a coupled simulation where fluid production causes a significant increase in the effective stress within a reservoir. Changing the in-situ rock stress state can alter the reservoir properties. As a result, porosity and permeability could be affected due to the rearrangement of rock particles and the redistribution of sensitive pore structures. It is known that permeability is more sensitive to stress changes than porosity.
Allergic diseases are characterized by a Th2 inflammatory response involving several
possible modulator factors (genetics and environmental factors), subject-related or antigenrelated
modulators such as adjuvants, solubility in the microenvironment of mucosa, size of
the sensitization agent, mucosa permeability, viral infections, and the greater or lesser
ability of effectors cells to liberate mediators.
Other factors include atmospheric pollution, exposure to tobacco, lifestyle-related diet and
hygiene habits and maternal effects.
2-Aminoethoxydiphenyl Borate (2-APB) has been extensively used recently as a membrane permeable modulator of inositol-1,4,5-trisphosphate-sensitive Ca2+ channels and store-operated Ca2+ entry. Here, we report that 2-APB is also an inhibitor of sarco/endoplasmic reticulum Ca2+ATPase (SERCA) Ca2+ pumps, and additionally increases ion leakage across the phospholipid bilayer. Therefore, we advise caution in the interpretation of results when used in Ca2+ signalling experiments.