The book Cell Interaction focuses on various processes that occur within and outside the cells. Cell interactions are important for functioning of many organ systems: cell adhesion, tissue development, cellular communication, inflammation, tumor metastasis, and microbial infection. Key features include developmental cell interactions, immune and neural cell interactions, cell interactions in normal and disease conditions and advanced level methods to evaluate cell interactions.
Alzheimer’s disease (AD) is an age-related, progressive degenerative dis-order that is characterized by synapse and neuron loss in the brain and the
accumulation of protein-containing deposits (referred to as ‘senile plaques’)
and neurofibrillary tangles. Insoluble amyloid b-peptide (Ab) fibrillar
aggregates found in extracellular plaques have long been thought to cause
the neurodegenerative cascades of AD.
Nhắc lại cấu trúc thần kinh của đường
dẫn truyền cảm giác đau. Điện thế màng tế
bào (điện thế nghỉ resting potential &
điện thế động )là thuộc tính sinh lý
quan trọng hàng đầu của neuron.
1.Toàn thể đường dẫn truyền cảm giác đau hướng tâm truyền 1.Tất cả các synapses của đường dẫn
truyền cảm giác đau là
synapse hóa (chemical synapse).
Drugs for the Suppression of Pain (Analgesics)
ing, or burning character, i.e., pain that can be localized only poorly. Impulse traffic in the neo- and paleospinothalamic pathways is subject to modulation by descending projections that originate from the reticular formation and terminate at second-order neurons, at their synapses with first-order neurons, or at spinal segmental interneurons (descending antinociceptive system).
Estrogen acts as a proconvulsant in several animal models of epilepsy, including amygdalal
kindling and pentylenetetrazol administration in ovariectomized rats (Hom and
Buterbaugh, 1986). Estrogen induces the formation of new excitatory synapses in the CA1
region of the hippocampus; and further, this estrogenic induction involves activation of Nmethyl-
Daspartate (NMDA) receptors (McEwen, 2002). Increasing the complexity of
hippocampal synaptic density is likely a mechanism for the proconvulsant activity of