Cerebral small

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  • Figure 27-3 Evidence of left hemispatial neglect and simultanagnosia. A. A 47-yearold man with a large frontoparietal lesion in the right hemisphere was asked to circle all the As. Only targets on the right are circled. This is a manifestation of left hemispatial neglect. B. A 70-year-old woman with a 2-year history of degenerative dementia was able to circle most of the small targets but ignored the larger ones. This is a manifestation of simultanagnosia.Cerebrovascular lesions and neoplasms in the right hemisphere are the most common causes of hemispatial neglect.

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  • In Chapter 5, Carrascosa Romero and de Cabo de la Vega consider the relevance of genetics and brain imaging findings in the diagnosis of diseases which result from abnormal development or neoplasia of cells derived from the neural crest (neurocristopathies). Koga and Saito (Chapter 6) provide a beautifully illustrated discussion of the use of neuroimaging tools to guide gamma knife radiosurgery.

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  • Hemiparesis Hemiparesis results from an upper motor neuron lesion above the midcervical spinal cord; most such lesions are above the foramen magnum. The presence of other neurologic deficits helps to localize the lesion. Thus, language disorders, cortical sensory disturbances, cognitive abnormalities, disorders of visual-spatial integration, apraxia, or seizures point to a cortical lesion. Homonymous visual field defects reflect either a cortical or a subcortical hemispheric lesion.

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  • Immunoblotting analyses revealed the presence of carb-oxylesterase in theporcine small intestine, liver, submaxillary andparotidglands, kidney cortex, lungs and cerebral cortex. In the intestinal mucosa, the pI 5.1 enzyme was detected in several subcellular fractions including the microvillar frac-tion. Both fatty monoacylated and diacylated monomeric (F1), trimeric (F3) and tetrameric (F4) forms of the intestinal protein were puri®ed here for the ®rst time by performing hydrophobic chromatography and gel ®ltration....

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  • MicroRNAs are small RNAs that attenuate protein expression by comple-mentary binding to the 3¢-UTR of a target mRNA. Currently, very little is known about microRNAs after cerebral ischemia. In particular, micro-RNA-21 (miR-21) is a strong antiapoptotic factor in some biological systems. We investigated the role of miR-21 after stroke in the rat.

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