Disease pathogenesis

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  • Autoimmune disease represents a group of more than 60 different chronic autoimmune diseases that affect approximately 6% of the population. It is the third major category of illness in the United States and many industrialized countries, following heart disease and cancer. Autoimmune diseases arise when one’s immune system actively targets and destroys self tissue resulting in clinical disease. Common examples include Systemic Lupus Erythematosus, Type 1 Diabetes, Rheumatoid Arthritis and Multiple Sclerosis.

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  • Celiac Disease (CD) or Gluten Sensitive Enteropathy (GSE) is a life‐long disorder. It is characterized by inflammation in the small intestine of genetically predisposed individuals caused by inappropriate immune response to gluten, a protein enriched in some of our common grains (wheat, rye and barley). The toxicity of gluten is manifested by the autoimmune action of T‐lymphocytes on mucosal cells in the small intestine, disrupting its vital function of absorbing nutrients from food.

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  • Tuyển tập các báo cáo nghiên cứu khoa học ngành y học tạp chí Medical Sciences dành cho các bạn sinh viên ngành y tham khảo đề tài: Vgf is a novel biomarker associated with muscle weakness in amyotrophic lateral sclerosis (ALS), with a potential role in disease pathogenesis...

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  • Tham khảo luận văn - đề án 'báo cáo y học: " vgf is a novel biomarker associated with muscle weakness in amyotrophic lateral sclerosis (als), with a potential role in disease pathogenesis"', luận văn - báo cáo phục vụ nhu cầu học tập, nghiên cứu và làm việc hiệu quả

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  • Tuyển tập các báo cáo nghiên cứu về y học được đăng trên tạp chí y học Critical Care giúp cho các bạn có thêm kiến thức về ngành y học đề tài: Infectious Disease: Pathogenesis, Prevention, and Case Studies...

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  • Tuyển tập báo cáo các nghiên cứu khoa học quốc tế ngành hóa học dành cho các bạn yêu hóa học tham khảo đề tài: The contribution of activated astrocytes to Ab production: Implications for Alzheimer’s disease pathogenesis

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  • There is great difficulty, in the case of a subject so large and complex as is disease, in giving a definition which will be accurate and comprehensive. Disease may be defined as "A change produced in living things in consequence of which they are no longer in harmony with their environment." It is evident that this conception of disease is inseparable from the idea of life, since only a living thing can become diseased.

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  • The subject of preventive medicine is one that is attracting world-wide attention to-day. We can hardly pick up a newspaper or magazine without seeing the subject discussed in some of its phases, and during the last few years several books have appeared devoted wholly or in part to the ways of preventing rather than curing many of our ills. Looking over the titles of these articles and books the reader will at once be impressed with the importance that is being given to the subject of the relation of insects to some of our common diseases.

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  • Part 1 book “Thyroid diseases - Pathogenesis, diagnosis, and treatment” has contents: Regulation of thyroid function, synthesis, and function of thyroid hormones, tests of thyroid function, thyroid autoantibodies, nonisotopic thyroid imaging, nontoxic goiter, thyroid nodule, acute and subacute thyroiditis,… and other contents.

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  • Part 2 book “Thyroid diseases - Pathogenesis, diagnosis, and treatment” has contents: Central hypothyroidism, diagnosis and treatment of hypothyroidism, toxic adenoma and multinodular toxic goiter, medullary carcinoma, anaplastic and other forms of thyroid carcinoma, non- thyroidal illness,… and other contents.

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  • Use of Fungi for Insect Control - Issues, Developments & Research Needs Entomopathogenic Fungi • Classification • Pathogenesis • Why fungi as BCA’s? • Commercialization • Registration • Some examples • Research Agriculture and Agriculture et Agri-Food Canada Agroalimentaire Canada

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  • Central to the development of glomerular inflammation and injury are alterations and abnormalities of various cytokines and signaling systems. There are four chapters in this book that deal with these aspects in the pathogenesis. The role of TGF-β in progressive glomerular disease is discussed in great detail in a chapter well written by Hyun Soon Lee, with particular reference to mesangial matrix accumulation, while the role of STAT3 activation in glomerulonephritis is elaborated in the well written chapter by Fumio Tsuji et al.

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  • Harrison's Internal Medicine Chapter 114. Molecular Mechanisms of Microbial Pathogenesis Molecular Mechanisms of Microbial Pathogenesis: Introduction Over the past three decades, molecular studies of the pathogenesis of microorganisms have yielded an explosion of information about the various microbial and host molecules that contribute to the processes of infection and disease.

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  • Encounters with Epithelial Cells Over the past decade, many bacterial pathogens have been shown to enter epithelial cells (Fig. 114-2); the bacteria often use specialized surface structures that bind to receptors, with consequent internalization. However, the exact role and the importance of this process in infection and disease are not well defined for most of these pathogens. Bacterial entry into host epithelial cells is seen as a means for dissemination to adjacent or deeper tissues or as a route to sanctuary to avoid ingestion and killing by professional phagocytes.

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  • Encounters with Phagocytes Phagocytosis and Inflammation Phagocytosis of microbes is a major innate host defense that limits the growth and spread of pathogens. Phagocytes appear rapidly at sites of infection in conjunction with the initiation of inflammation. Ingestion of microbes by both tissue-fixed macrophages and migrating phagocytes probably accounts for the limited ability of most microbial agents to cause disease.

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  • Invasion Many diseases are caused primarily by pathogens growing in tissue sites that are normally sterile. Pneumococcal pneumonia is mostly attributable to the growth of S. pneumoniae in the lung and the attendant host inflammatory response, although specific factors that enhance this process (e.g., pneumolysin) may be responsible for some of the pathogenic potential of the pneumococcus. Disease that follows bacteremia and invasion of the meninges by meningitisproducing bacteria such as N. meningitidis, H. influenzae, E.

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  • Transmission to New Hosts As part of the pathogenic process, most microbes are shed from the host, often in a form infectious for susceptible individuals. However, the rate of transmissibility may not necessarily be high, even if the disease is severe in the infected individual, as transmissibility and virulence are not linked traits.

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  • The etiology of PV is unknown. Although nonrandom chromosome abnormalities such as 20q, trisomy 8, and especially 9p, have been documented in up to 30% of untreated PV patients, unlike CML no consistent cytogenetic abnormality has been associated with the disorder. However, a mutation in the autoinhibitory, pseudokinase domain of the tyrosine kinase JAK2—which replaces valine with phenylalanine (V617F), causing constitutive activation of the kinase— appears to have a central role in the pathogenesis of PV.

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  • The human body as a mechanism is far from perfect. It can be beaten or surpassed at almost every point by some product of the machine-shop or some animal. It does almost nothing perfectly or with absolute precision. As Huxley most unexpectedly remarked a score of years ago, "If a manufacturer of optical instruments were to hand us for laboratory use an instrument so full of defects and imperfections as the human eye, we should promptly decline to accept it and return it to him. But," as he went on to say, "while the eye is inaccurate as a microscope, imperfect as a telescope, crude...

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  • Background: Dercum’s disease (DD) is characterised by obesity and chronic pain ( 3 months) in the adipose tissue. The pathogenesis of DD is unknown, but inflammatory components have been proposed. In previous reports and studies, an inconsistent picture of the histological appearance of the adipose tissue in DD has been described. The aim of this investigation was to examine the histological appearance of adipose tissue in patients with DD, with particular focus on inflammatory signs. Methods: Fat biopsies were sampled from painful regions from 53 patients with DD.

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