Chromosome evolution

Tuyển tập báo cáo các nghiên cứu khoa học quốc tế ngành y học dành cho các bạn tham khảo đề tài: Dynamic distribution patterns of ribosomal DNA and chromosomal evolution in Paphiopedilum, a lady’s slipper orchid

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Tuyển tập các báo cáo nghiên cứu về y học được đăng trên tạp chí y học Minireview cung cấp cho các bạn kiến thức về ngành y đề tài: Is mammalian chromosomal evolution driven by regions of genome fragility?

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Jeffrey A Bailey*, Robert Baertsch†, W James Kent†, David Haussler‡ and Evan E Eichler* Addresses: *Department of Genetics, Center for Computational Genomics, Case Western Reserve University School of Medicine and University Hospitals of Cleveland, Cleveland, OH 44106, USA. †Center for Biomolecular Science and Engineering, University of California, Santa Cruz, CA 95064, USA. ‡Howard Hughes Medical Institute, 321 Baskin Engineering, University of California, Santa Cruz, CA 95064, USA. Correspondence: Evan E Eichler. E-mail: eee@cwru.

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Chapter 13 - Chromosomal rearrangements and changes in chromosome number. This chapter presents the following content: Rearrangements of DNA sequences, transposable genetic elements, rearrangements and evolution: a speculative comprehensive example, changes in chromosome number, emergent technologies: beyond the karyotype.

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Genetics is the science of genes, heredity, and the variation of organisms.[37][38] Genes encode the information necessary for synthesizing proteins, which in turn play a large role in influencing (though, in many instances, not completely determining) the final phenotype of the organism. In modern research, genetics provides important tools in the investigation of the function of a particular gene, or the analysis of genetic interactions. Within organisms, genetic information generally is carried in chromosomes, where it is represented in the chemical structure of particular DNA molecules....

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Tuyển tập các báo cáo nghiên cứu về sinh học được đăng trên tạp chí sinh học thế giới đề tài: Chromosomal mapping, differential origin and evolution of the S100 gene family

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Uncovering Recurrent Submicroscopic Rearrangements As a Cause of Disease For five decades since Fred Sanger's (1) seminal discovery that proteins have a specific structure, since Linus Pauling's (2) discovery that hemoglobin from patients with sickle cell anemia is molecularly distinct, and since Watson and Crick's (3) elucidation of the chemical basis of heredity, the molecular basis of disease has been addressed in the context of how mutations affect the structure, function, or regulation of a gene or its protein product.

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Although DNA from any two people is more alike than different, many chromosome regions exhibit sequence differences between individuals. Such variable sequences are termed “polymorphic” (meaning many forms) and are used in the study of human evolution, as well as for disease and identity testing. Many polymorphisms are located in the estimated 98% of the human genome that does not encode protein. This experiment examines a polymorphism in the human genome that is caused by the insertion of an Alu transposon, or transposable element.

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