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Loss of consciousness

Xem 1-19 trên 19 kết quả Loss of consciousness
  • Subarachnoid hemorrhage (SAH) is sudden bleeding into the subarachnoid space. The most common cause of spontaneous SAH is a ruptured aneurysm. Symptoms include sudden, severe headache, usually with loss or impairment of consciousness. It has a lot of dangerous complications especially re-bleeding and vasospasm which are the major causes of death and disability. Objective: To study on clinical characteristics, computed tomography brain imaging, causes and complications of subarachnoid hemorrhage.

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  • The quality of life after rapid bone loss among individuals is one of the key health issues today, which might be due to hormonal factors and lack of proper nutrition that lead to an increased risk of fractures. The experimental data suggested a significant potential of Consciousness Energy Healing based vitamin D3 and DMEM medium on various bone health parameters.

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  • Ketamine is a frequently used intravenous anesthetic, which can reversibly induce loss of consciousness (LOC). Previous studies have demonstrated that thalamocortical system is critical for information transmission and integration in the brain. The ventral posteromedial nucleus (VPM) is a critical component of thalamocortical system.

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  • The α2-adrenergic agonist dexmedetomidine (DEX) is a sedative and can be used as an adjunct to hypnotics. The study sought to evaluate the effects of different doses of DEX on the requirements for propofol for loss of consciousness (LOC) in patients monitored via the bispectral index (BIS).

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  • Maternal cardiac arrest during cesarean section (CS) is an extremely rare but devastating complication. Preventing emergency events from developing into maternal cardiac arrest is one of the most challenging clinical scenarios.

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  • Highly structured electroencephalography (EEG) oscillations can occur in adults during etomidateinduced general anesthesia, but the link between these two phenomena is poorly understood. Therefore, in the present study, we investigated the electroencephalogram dynamics of etomidate-induced loss of consciousness (LOC) in order to understand the neurological mechanism of etomidate-induced LOC.

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  • part 2 present the content: dysphagia; dyspnoea; fatigue; fever; gastrointestinal haemorrhage: haematemesis and rectal bleeding; haematuria; haemoptysis; headache; jaundice; joint swelling; leg swelling; limb weakness; low back pain; nausea and vomiting; transient loss of consciousness: syncope and seizures...

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  • We need to understand our impacts on the natural world in the fight against species loss and climate change. We need to change our patterns of consumption to combat climate change, conserve the Amazon rainforest and protect our oceans. We must become active citizens and conscious consumers, aware of the consequences of our actions and the purchases we make....

    pdf326p doidieumuonnoi 11-06-2013 50 6   Download

  • Availability of fossil fuels became readily and abundantly available during the mid to latter part of the 20th century, and building design responded with mechanical and electrical systems that in many instances consumed the fuel excessively. Large single glazed areas caused over-heating, glare and solar discomfort in the summer, combined with cold draughts and high heat losses in the winter. Thermostatic control was often rudimentary and compensated by opening and closing windows accordingly.

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  • The recent crisis was the result of a cascade of shocks that originated in the financial sector. It makes sense, therefore, to start by asking how the structure of the banking sector affected outcomes across countries. Deposits are thought to be a relatively stable source of bank funding; economies where banks have relatively low loan-to-deposit ratios before the beginning of the crisis may therefore be relatively robust. Similarly, better capitalised banks should be better able to absorb losses while maintaining the supply of funding to support the real economy.

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  • For the last nineteen years, I have had the great pleasure and privilege of teaching and learning about healing at the Rice Diet Program in Durham, North Carolina. Participants here, fondly nicknamed Ricers, have the opportunity to experience the fastest, safest, and most effective way to lose weight and improve every other modifi able risk factor of heart disease and, in fact, most diseases.

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  • Communities in crisis suffer a loss of access to services, which UNFPA works to restore or to provide in temporary locations. While access is most obviously a problem for refugees and the internally displaced, people in surrounding communities and other family members may also be affected by the crisis. Access continues to be a priority once a crisis concludes, during the process of recovery and rehabilitation.

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  • Presyncopal symptoms vary in duration and may increase in severity until loss of consciousness occurs, or they may resolve prior to loss of consciousness if the cerebral ischemia is corrected. The differentiation of syncope from seizure is an important, sometimes difficult, diagnostic problem.Syncope may be benign when it occurs as a result of normal cardiovascular reflex effects on heart rate and vascular tone, or serious when due to a life-threatening cardiac arrhythmia.

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  • Anaphylactic Reaction This severe reaction presents after transfusion of only a few milliliters of the blood component. Symptoms and signs include difficulty breathing, coughing, nausea and vomiting, hypotension, bronchospasm, loss of consciousness, respiratory arrest, and shock. Treatment includes stopping the transfusion, maintaining vascular access, and administering epinephrine (0.5–1.0 mL of 1:1000 dilution subcutaneously). Glucocorticoids may be required in severe cases.

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  • Harrison's Internal Medicine Chapter 25. Numbness, Tingling, and Sensory Loss Numbness, Tingling, and Sensory Loss: Introduction Normal somatic sensation reflects a continuous monitoring process, little of which reaches consciousness under ordinary conditions. By contrast, disordered sensation, particularly when experienced as painful, is alarming and dominates the sufferer's attention. Physicians should be able to recognize abnormal sensations by how they are described, know their type and likely site of origin, and understand their implications. Pain is considered separately in Chap.

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  • Syncope: Treatment The treatment of syncope is directed at the underlying cause. This discussion will focus on disorders of autonomic control. Arrhythmias are discussed in Chaps. 225 and 226, valvular heart diseases in Chap. 230, and cerebrovascular disorders in Chap. 364.Certain precautions should be taken regardless of the cause of syncope. At the first sign of symptoms, patients should make every effort to avoid injury should they lose consciousness.

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  • Glossopharyngeal Neuralgia Syncope due to glossopharyngeal neuralgia (Chap. 371) is preceded by pain in the oropharynx, tonsillar fossa, or tongue. Loss of consciousness is usually associated with asystole rather than vasodilatation. The mechanism is thought to involve activation of afferent impulses in the glossopharyngeal nerve that terminate in the nucleus solitarius of the medulla and, via collaterals, activate the dorsal motor nucleus of the vagus nerve.

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  • Harrison's Internal Medicine Chapter 21. Syncope Syncope: Introduction Syncope, a transient loss of consciousness and postural tone due to reduced cerebral blood flow, is associated with spontaneous recovery. It may occur suddenly, without warning, or may be preceded by symptoms of faintness ("presyncope"). These symptoms include lightheadedness, dizziness, a feeling of warmth, diaphoresis, nausea, and visual blurring occasionally proceeding to transient blindness.

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  • Overdose, including self-poisoning, causes bradycardia, heart block, hypotension and low output cardiac failure that can proceed to cardiogenic shock; death is more likely with agents having membrane stabilising action (see Table 23.1). Bronchoconstriction can be severe, even fatal, in patients subject to any bronchospastic disease; loss of consciousness may occur with lipid-soluble agents that penetrate the central nervous system. Receptor blockade will outlast the persistence of the drug in the plasma. Rational treatment includes: • Atropine (1-2 mg i.v.

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