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Tongue lesions
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Ebook Concise oral medicine: Part 1 includes content: Ulcerative and vesiculobullous lesions, red and white lesions, gingival enlargement, tongue lesions, granulomatous diseases and STDs, cross-infection control,… and other contents.
173p
longtimenosee03
01-02-2024
2
0
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Patients with tongue cancer frequently show loss of heterozygosity (LOH) of the von Hippel–Lindau (VHL) tumor suppressor gene. However, expression of VHL protein (pVHL) in tongue cancer has rarely been investigated and remains largely unknown.
8p
vidhaka2711
31-07-2020
10
1
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(bq) part 2 book “cawson’s essentials of oral pathology and oral medicine” has contents: tongue disorders, benign chronic white mucosal lesions, oral premalignancy, common benign mucosal swellings, immunodefi ciencies and hiv disease,… and other contents.
264p
tieu_vu15
07-09-2018
45
3
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Oral lesions were found in 61 (78.2%) cases. Sites involved were inner aspect of the lips, gums, buccal mucosa, tongue and the hard palate. Small aphthous like lesions measuring 1 to 3 mm were the usual mucosal presentation. Most common systemic symptoms (Table II) were fever and anorexia. History of mild fever either preceding to or simultaneously with the eruption was present in 58 (74.3%) cases. Fever appeared on the same day in 70% cases and 1 day before onset of rash in 30% cases. Fever persisted for 1 to 2 days following onset. Sore throat was a symptom during...
9p
connicquy
14-12-2012
80
3
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Pink Lesions The cutaneous lesions associated with primary systemic amyloidosis are often pink in color and translucent. Common locations are the face, especially the periorbital and perioral regions, and flexural areas. On biopsy, homogeneous deposits of amyloid are seen in the dermis and in the walls of blood vessels; the latter lead to an increase in vessel wall fragility. As a result, petechiae and purpura develop in clinically normal skin as well as in lesional skin following minor trauma, hence the term pinch purpura.
5p
konheokonmummim
30-11-2010
93
4
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Dermatologic Diseases See Tables 32-1, 32-2, and 32-3 and Chaps. 52, 53, 54, 55, and 56.
5p
ongxaemnumber1
29-11-2010
88
5
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Pathogenesis Upper Motor Neuron Weakness This pattern of weakness results from disorders that affect the upper motor neurons or their axons in the cerebral cortex, subcortical white matter, internal capsule, brainstem, or spinal cord (Fig. 23-1). Such lesions produce weakness through decreased activation of the lower motor neurons. In general, distal muscle groups are affected more severely than proximal ones, and axial movements are spared unless the lesion is severe and bilateral.
5p
ongxaemnumber1
29-11-2010
68
2
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