Venous malformation

Part 1 book "Plastic surgery - Clinical problem solving" includes content: Encephalocele, coronal synostosis, sagittal synostosis, scalp wound, eyelid ptosis, upper eyelid defect, ectropion, lower eyelid lesion, subjective nasal deformity, nasal reconstruction, unilateral cleft lip, bilateral cleft lip, upper lip lesion, lower lip reconstruction, cleft palate, micrognathia, treacher collins syndrome, prominent ear deformity, microtia, ear lesion, venous malformation, panfacial fracture, cheek mass, lesion of the cheek,... and other contents.

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Cavernous venous malformation is an uncommon entity that occurs in around 0.5% of the general population. Cerebellar cavernous venous malformation accounts for 1.2–11.8% of intracranial cavernous venous malformation cases. Patients are commonly asymptomatic until a hemorrhage occurs.

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The term hepatopulmonary syndrome typically applies to cyanosis that results from “intrapulmonary vascular dilatation” due to advanced liver disease. Similar findings may result from a congenital portosystemic shunt without liver disease.

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Klippel-Trenaunay syndrome (KTS) is a rare congenital syndrome characterized by the triad of venous varicosities, capillary malformations and limb hypertrophy. However, KTS may rarely occur in combination with kyphoscoliosis.

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Klippel-Trenaunay syndrome (KTS) is a rare complex vessel malformation syndrome characterized by venous varicosities, capillary malformations, and limb hypertrophy. However, extensive heterotopic ossification (HO) secondary to this syndrome is extremely rare.

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Klippel-Trénaunay syndrome (KTS) is a rare congenital condition characterized by the clinical triad of capillary malformations (port wine stains), varicose veins with or without venous malformations, and bony and/or soft tissue hypertrophy.

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A TIE2 mutation causing arginine-to-tryptophan substitution at residue 849 (TIE2-R849W) is commonly identified in heredofamilial venous malformation. However, there is no in vivo model to confirm the pathogenic role of TIE2-R849W. Humanized TIE2-R849W plasmid was constructed via PCR-mediated site-directed mutagenesis.

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part 2 book “handbook of cerebrovascular disease and neurointerventional technique” has contents: intracranial aneurysms and subarachnoid hemorrhage, arteriovenous malformations, dural arteriovenous fistulas, cavernous malformations and venous angiomas, extracranial cerebrovascular occlusive disease,… and other contents.

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part 2 book “vascular surgery principles and practice” has contents: cerebrovascular disease, visceral arterial disease, vascular disorders of the upper extremity and vasculitis, venous and lymphatic disorders, vascular trauma, compartment syndrome, vascular access, malformations and transplantation, surgical techniques.

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Blue Lesions Lesions that are blue in color are the result of either vascular ectasias and tumors or melanin pigment in the dermis. Venous lakes (ectasias) are compressible dark-blue lesions that are found commonly in the head and neck region. Venous malformations are also compressible blue papulonodules and plaques that can occur anywhere on the body, including the oral mucosa. When there are multiple rather than single congenital lesions, the patient may have the blue rubber bleb syndrome or Mafucci's syndrome.

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Table 34-1 Differential Diagnosis of Hemoptysis Source other than the lower respiratory tract Upper airway (nasopharyngeal) bleeding Gastrointestinal bleeding Tracheobronchial source Neoplasm (bronchogenic carcinoma, endobronchial metastatic tumor, Kaposi's sarcoma, bronchial carcinoid) Bronchitis (acute or chronic) Bronchiectasis Broncholithiasis Airway trauma Foreign body Pulmonary parenchymal source Lung abscess Pneumonia Tuberculosis Mycetoma ("fungus ball") Goodpasture's syndrome Idiopathic pulmonary hemosiderosis Wegener's granulomatosis Lupus pneumonitis Lung ...

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