Serum enzymes

Xem 1-19 trên 19 kết quả Serum enzymes
  • Previous biochemical studies have indicated that the membrane-bound thyrotropin-releasing hormone (TRH)-degrading enzyme (TRH-DE) from brain and liver and the serum TRH-DE are derived from the same gene. These studies also suggested that the serum enzyme is of liver origin. The present study was undertaken to verify these hypotheses. In different species, a close relationship between the activities of the serum enzyme and the particulate liver enzyme was noticed.

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  • Back or flank pain is a critical symptom that indicates a retrograde infection to kidneys, risk factor for which is graft versus host disease. Furthermore, adenovirus-induced nephritis is a high risk factor for disseminated adenovirus infection including symptomatic adenoviremia (Bruno et al., 2004). Clinical presentation of adenoviremia is low to high grade fever refractory to antibiotic or antifungal agents followed by multiple organ failure including acute renal failure which presents oliguria, elevation of serum creatinine and enlarged kidneys with hypo-density in CT scan.

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  • Human serum paraoxonase 1 (HuPON1; EC is a calcium-depend-ent six-foldb-propeller enzyme that has been shown to hydrolyze an array of substrates, including organophosphorus (OP) chemical warfare nerve agents. Although recent efforts utilizing site-directed mutagenesis have demonstrated specific residues (such as Phe222 and His115) to be import-

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  • Tuyển tập các báo cáo nghiên cứu khoa học quốc tế về bệnh thú y đề tài: Comparison of an enzyme-linked immunosorbent assay with serum neutralization test for serodiagnosis of porcine epidemic diarrhea virus infection

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  • Angina is the characteristic pain of IHD. It is caused by atherosclerosis leading to stenosis (partial occlusion) of one or more coronary arteries. Patients with chronic stable angina have an average annual mortality of 2 percent or less. Acute myocardial infarction (AMI) is the total occlusion of a major coronary artery with a complete lack of oxygen and nutrients leading to cardiac muscle necrosis. AMI is usually diagnosed by changes in the electrocardiogram; by elevated serum enzymes, such as creatine phosphokinase and troponin T or I; and by pain similar to that of angina.

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  • Hepatocytes of the periportal and perivenous zones of the liver lobule show marked differences in the contents and activities of many enzymes and other proteins. Previous studies from our and other groups have pointed towards an important role of b-catenin-dependent signaling in the regula-tion of expression of genes encoding proteins with preferential perivenous localization, whereas, in contrast, signaling through Ras-dependent path-way(s) may induce a ‘periportal’ phenotype.

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  • The functional site ofÔphospholipase A2 inhibitor from pythonÕ (PIP) was predicted based on the hypothesis of proline brackets. Using di€erent sources of secretory phospholipase A2(sPLA2 s) as enzyme, and [ 3 H]arachido-nate-labelledEscherichia colias substrate, short synthetic peptides representing the proposed site were examined for their secretoryphospholipase A2(sPLA2 ) inhibitoryactivity.

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  • (BQ) Part 1 book "Tietz fundamentals of clinical chemistry and molecular diagnostics" presents the following contents: Principles of laboratory medicine, analytical techniques and instrumentation, analytes. Invite you to consult.

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  • GOT, GPT là 2 enzym trao đổi amin (transaminase), có nhiều ở các tổ chức của cơ thể. Trong các enzym trao đổi amin, GOT và GPT có hoạt độ cao hơn cả và có ứng dụng nhiều trong lâm sàng. SGOT: Serum Glutamic Oxaloacetic Transaminase. Men này chủ yếu ở các mô có chuyển hóa cao như gan, tim, cơ xương. Chỉ số men tăng trong các trường hợp tổn thương tế bào gan do viêm, xơ, ung thư; Tổn thương tim do nhồi máu…Giảm trong một số trường hợp như tiểu đường, thai kỳ, Beriberi… ...

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  • Một số nguyên nhân hay gặp làm tăng nồng độ men gan: bao gồm những nguyên nhân tại gan và nguyên nhân ngoài gan như: Men gan là gì ? Men gan là một loại enzyme bình thường nằm trong tế bào gan, tham gia vào quá trình chuyển hóa các chất trong cơ thể. Có bao nhiêu loại men gan? – AST (Aspartate Transaminase) hay SGOT (Serum Glutamic Oxaloacetic Transaminase) hay ASAT (Aspartate AminoTransferase) là một enzyme ở bào tương và ty thể, hiện diện ở tế bào gan, tim, cơ vân, thận, não và tụy.

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  • Deferasirox is a promising oral iron-chelating agent. Single daily doses of 20 or 30 mg deferasirox produced reductions in liver iron concentration comparable to desferoxamine in chronically transfused adult and pediatric patients. Deferasirox produces some elevations in liver enzymes and slight but persistent increases in serum creatinine, without apparent clinical consequence. Other toxicities are similar to those of desferoxamine. Its toxicity profile is acceptable, although long-term effects are still being evaluated.

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  • The complement system (Chap. 308) consists of a group of serum proteins functioning as a cooperative, self-regulating cascade of enzymes that adhere to— and in some cases disrupt—the surface of invading organisms. Some of these surface-adherent proteins (e.g., C3b) can then act as opsonins for destruction of microbes by phagocytes. The later, "terminal" components (C7, C8, and C9) can directly kill some bacterial invaders (notably, many of the neisseriae) by forming a membrane attack complex and disrupting the integrity of the bacterial membrane, thus causing bacteriolysis.

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  • An underlying maternal folate metabolic abnormality has also been postulated. One abnormality has been identified: reduced activity of the enzyme 5,10-methylene-THF reductase (MTHFR) (Fig. 100-1) caused by a common 677CÆT polymorphism in the MTHFR gene. In one study, the prevalence of this polymorphism was found to be higher in the parents of NTD fetuses and in the fetuses themselves: homozygosity for the TT mutation was found in 13% compared with 5% in control subjects. The polymorphism codes for a thermolabile form of MTHFR.

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  • Diagnostic Tests The choice of diagnostic tests should be guided by the history and the physical examination. Measurements of serum electrolytes, glucose, and the hematocrit are usually indicated. Cardiac enzymes should be evaluated if myocardial ischemia is suspected. Blood and urine toxicology screens may reveal the presence of alcohol or other drugs. In patients with possible adrenocortical insufficiency, plasma aldosterone and mineralocorticoid levels should be obtained.

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  • Hemolytic disorders that cause excessive heme production may be either inherited or acquired. Inherited disorders include spherocytosis, sickle cell anemia, thalassemia, and deficiency of red cell enzymes such as pyruvate kinase and glucose-6-phosphate dehydrogenase. In these conditions, the serum bilirubin rarely exceeds 86 µmol/L (5 mg/dL). Higher levels may occur when there is coexistent renal or hepatocellular dysfunction or in acute hemolysis such as a sickle cell crisis.

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  • Laboratory Testing for HIT HIT (antiheparin/PF4) antibodies can be detected using two types of assays. The most widely available is an enzyme-linked immunoassay (ELISA) with PF4/polyanion complex as the antigen. Since many patients develop antibodies but do not develop clinical HIT, the test has a low specificity for the diagnosis of HIT. This is especially true in patients who have undergone cardiopulmonary bypass surgery, where approximately 50% of patients develop these antibodies postoperatively.

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  • The interaction of xenon with copper⁄6-hydroxydopa (2,4,5-trihydroxy-phenethylamine) quinone (TPQ) amine oxidases from the plant pulses lentil (Lens esculenta) and pea (Pisum sativum) (seedlings), the perennial Mediter-ranean shrub Euphorbia characias(latex), and the mammals cattle (serum) and pigs (kidney), were investigated by NMR and optical spectroscopy of the aqueous solutions of the enzymes.

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  • Loss of E-cadherin-mediated cell–cell adhesion and expression of proteolytic enzymes characterize the transition from benign lesions to invasive, metastatic tumor, a rate-limiting step in the progression from adenoma to carcinoma in vivo. A soluble E-cadherin fragment found recently in the serum and urine of cancer patients has been shown to disrupt cell–cell adhe-sion and to drive cell invasion in a dominant-interfering manner.

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  • For bovine serum amine oxidase, two different mechanisms of substrate-induced inactivation have been proposed. One consists of a slow oxidation by H2O2of a conserved residue in the reduced enzyme after the fast turnover phase [Pietr-angeli, P., Nocera, S., Fattibene, P., Wang, X.T., Mondovı`, B. &Morpurgo, L. (2000)Biochem. Biophys. Res. Commun. 267, 174–178] and the other of the oxidation byH2O2of the dihydrobenzoxazole in equilibrium with the product Schiff base, during the catalytic cycle [Lee, Y., Shepard, E., Smith, J., Dooley, D.M. & Sayre, L.M. (2001)Biochemistry40, 822–829]. ...

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