Chapter 101. Hemolytic Anemias and Anemia Due to Acute Blood Loss (Part 11)

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Table 101-5 Drugs that Carry Risk of Clinical Hemolysis in Persons with G6PD Deficiency Definite Risk Risk Possible Risk Doubtful Antimalarials Primaquine uine Chloroq Quinine Dapsone/chlorp roguanil Sulphonamides/s ulphones ole Sulphametoxaz zine Sulfasala ole Sulfisoxaz Others idine Sulfadim ne Sulfadiazi Dapsone Antibacterial/anti biotics Cotrimoxazole xacin Ciproflo Chloramp henicol Nalidixic acid cin Norfloxa pAminosalicylic acid Nitrofurantoin Niridazole Antipyretic/analg esics Acetanilide Acetylsa Acetylsali licylic acid high cylic acid 3 g/d) Phenazopyridin e (Pyridium) phen Acetamino Phenacetin Other Naphthalene Vitamin K analogues in Doxorubic Methylene blue Ascorbic acid 1 g d Probeneci Rasburic ase Table 101-6 Diseases/Clinical Situations with Predominantly Intravascular Hemolysis Onset/ Main riate Approp Comme ...

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Nội dung Text: Chapter 101. Hemolytic Anemias and Anemia Due to Acute Blood Loss (Part 11)

Chapter 101. Hemolytic Anemias and Anemia Due to Acute Blood Loss (Part 11) Table 101-5 Drugs that Carry Risk of Clinical Hemolysis in Persons with G6PD Deficiency Definite Risk Possible Doubtful Risk Risk Antimalarials Primaquine Chloroq Quinine uine Dapsone/chlorp roguanil Sulphonamides/s Sulphametoxaz Sulfasala Sulfisoxaz ulphones ole zine ole
Others Sulfadim Sulfadiazi idine ne Dapsone Antibacterial/anti Cotrimoxazole Ciproflo Chloramp biotics xacin henicol Nalidixic acid Norfloxa p- cin Aminosalicylic acid Nitrofurantoin Niridazole Antipyretic/analg Acetanilide Acetylsa Acetylsali esics licylic acid high cylic acid 3 g/d)
Phenazopyridin Acetamino e (Pyridium) phen Phenacetin Other Naphthalene Vitamin Doxorubic K analogues in Methylene blue Ascorbic Probeneci acid >1 g d Rasburic ase Table 101-6 Diseases/Clinical Situations with Predominantly Intravascular Hemolysis Onset/ Main Approp Comme riate
Time Course Mechanism Diagnostic nts Procedure Mismatche Abrupt Nearly Repeat d blood always ABO cross match transfusion incompatibility Paroxysmal Chronic Comple Flow Exacerba nocturnal with acute ment (C)- cytometry to tions due to C hemoglobinuria exacerbations mediated display a activation (PNH) destruction of CD59(–) red through any CD59(–) red cell population pathway cells Paroxysmal Acute Immune Test for Often cold lysis of normal Donath- triggered by hemoglobinuria red cells Landsteiner viral infection (PCH) antibody Septicemia Very Exotoxi Blood Other acute ns produced by cultures organisms may
Clostridium be responsible perfringens Microangio Acute Red cell Red cell Different pathic or chronic fragmentation morphology on causes ranging blood smear from endothelial damage to hemangioma to leaky prosthetic heart valve March Abrupt Mechan Targete hemoglobinuria ical destruction d history taking Favism Acute Destruc G6PD Triggere tion of older assay d by ingestion fraction of of large dish of G6PD- fava beans; but deficient red trigger can be
cells infection or drug instead